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PII: S0893-133X(01)00226-3 - Nature The objective of this study was to establish the effects of prefrontocortical dopamine depletion on opiate withdrawal and prefrontocortical neurochemical changes elicited by morphine dependence
Periaqueductal Gray and Rostromedial Tegmental Inhibitory . . . We describe differences in synaptic plasticity and behavior when optogenetically driving two opiate-sensitive GABAergic inputs to the VTA, the rostromedial tegmental nucleus (RMTg), and the periaqueductal gray (PAG)
Seasonal differences in the action of morphine and naloxone . . . Investigations of the membrane m chanisms of effects of agonists (morphine and enkepha-lins) and antagonists (naloxone) of opiate receptors have shown that opiates can not only act on themembrane potential of HeTix neurons [3, 4], but can also weaken the response of these neurons in a naloxone-dependent manner o serotonin [i] and dopamine [2
Dopaminergic Transmission Between Identified Neurons From the . . . dopamine to follower cells of RPeDl, in situ, mimicked the effects of RPeDl stimulation Dose- response curves were constructed for the excitatory effect of dopa- mine on follower cells, visceral dorsal two and three (VD2 3) (ED 5 = 39 PM; Hill coefficient = 1 03)) and the inhibitory effect of