COCOLOCO NIGHT CLUBBusiness Directories,Company Directories

Company Name: Corporate Name:	COCOLOCO NIGHT CLUB
Company Title:
Company Description:
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Company Address:	10018 105 St NW,EDMONTON,AB,Canada
ZIP Code: Postal Code:	T5J1C6
Telephone Number:	7804250933
Fax Number:	6045884337
Website:
Email:
USA SIC Code(Standard Industrial Classification Code):	581304
USA SIC Description:	Night Clubs
Number of Employees:	1 to 4
Sales Amount:	Less than $500,000
Credit History: Credit Report:	Unknown
Contact Person:
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Overcoming evasive resistance from vascular endothelial growth factor a . . .
In conclusion, sarcomas respond to increased hypoxia by expressing HIF-1α target genes that may promote resistance to antiangiogenic and other therapies HIF-1α inhibition blocks this evasive resistance and augments destruction of the tumor vasculature
Targeted inhibition of the HNF1A SHH axis by triptolide . . . - PubMed
Our results illuminate a novel molecular mechanism through which triptolide targets and inhibits HNF1A, thereby impeding the activation of the Hedgehog signaling pathway and reducing the expression of ABCB1
HNF1A regulates colorectal cancer progression and drug . . . - Nature
In conclusion, HNF1A expression can reflect resistance to anticancer drug treatment, and its suppression improves anticancer drug sensitivity as a new therapeutic target
Integrated bioinformatic analysis of HNF1A in human cancers
In this study, we first applied The Cancer Genome Atlas (TCGA) database to analyze the expression and mutations of HNF1A in different types of cancer Furthermore, we evaluated the association of HNF1A with the infiltration of immune cells and their prognostic value in patients with cancer
Regulatory mechanisms and potential medical applications of HNF1A-AS1 . . .
HNF1A-AS1 exerts its oncogenic roles through a variety of molecular mechanisms Moreover, aberrant HNF1A-AS1 expression is associated with diverse clinical features in cancer patients
Inhibition of vascular endothelial growth factor A and hypoxia . . .
HIF-1α inhibition is highly effective when combined with RT and VEGF-A inhibition in blocking sarcoma growth by maximizing DNA damage and apoptosis in tumor EC, leading to loss of tumor vasculature
Molecular mechanisms underpinning sarcomas and implications for current . . .
To identify novel treatment strategies for sarcomas, research in recent years has adopted a mechanism-based search for molecular markers for targeted therapy which has included recapitulating
Elevated HNF1A expression promotes radiation-resistance via driving . . .
Abstract Purpose: Radiotherapy is a major modality for treatment of local advanced esophageal squamous cell carcinoma (ESCC) Hepatocyte nuclear factor 1-alpha (HNF1A) is involved in regulation of tumor cell proliferation, apoptosis, cycle distribution, invasion metastasis and chemical resistance
Multimodal targeting of tumor vasculature and cancer stem-like cells in . . .
In this study, we hypothesized that the combination of VEGF-A inhibition and HIF-1α inhibition would attenuate tumor vasculature and enhance the effects of hypoxia-activated chemotherapy on sarcoma stem-like cells
Targeted inhibition of the HNF1A SHH axis by triptolide . . . - Nature
We demonstrated that triptolide directly bound to HNF1A, one of the transcription factors of SHH, and inhibited HNF1A SHH expression, ensuing in attenuation of Hedgehog signaling