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Kidney, Renal Tubule - Dilation - Nonneoplastic Lesion Atlas Secondary mechanisms of tubule dilation may result from lower urinary tract obstruction, the deposition of tubule crystals, interstitial inflammation and or fibrosis, and chronic progressive nephropathy (Figure 4) A few dilated tubules may be regarded as normal histologic variation
Interstitium tubules - kidneypathology. com In this chapter we review renal alterations in which tubules and or interstitium are the primary target of the pathogenic process
Chronic Interstitial Fibrosis and Tubular Atrophy - UPMC Tubular atrophy refers to the presence of tubules with thick redundant basement membranes, or a reduction of greater than 50% in tubular diameter compared to sorrounding non-atrophic tubules
Kidney Tubular and Interstitial Pathology Preview: Histological . . . Interstitium, f three basic categories: inflammatory involvement, ischemia vasoconstricton, toxic injury Tubule and or interstitial damage generally begins with leukocyte migration (type of leukocyte is indicat e of disease processes) to the site(s), inflammatio (acute) Chronic in Physiological condition(s):
Tubulointerstitial damage and progression of renal failure In addition, we present an overview of the pathways involved in the progression of tubulointerstitial fibrosis and a brief summary of the treatments used to retard the progression to end-stage renal failure
AJKD Atlas of Renal Pathology: Kidney Transplant Interstitial Fibrosis . . . In 2005, the Banff working group of transplant pathology intro-duced the term interstitial fibrosis tubular atrophy (IF TA), to emphasize that no etiology is assigned when this diagnosis is made Patients may be of any age IF TA typically develops months or even years after transplantation
Kidney, Renal Tubule - Dilation Secondary mechanisms of tubule dilation may result from lower urinary tract obstruction, the deposition of tubule crystals, interstitial inflammation and or fibrosis, and chronic progressive nephropathy (Figure 4)