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Homepage | Gut Gut is a leading international journal in gastroenterology and hepatology and has an established reputation for publishing first class clinical research of the alimentary tract, the liver, biliary tree and pancreas
Authors | Gut Editorial policies and guidelines for authors seeking to submit to Gut, a leading international journal in gastroenterology and hepatology
About | Gut Gut is a leading international journal in gastroenterology and hepatology and has an established reputation for publishing first class clinical research of the alimentary tract, the liver, biliary tree and pancreas
Gastrointestinal microbiota and inflammasomes interplay in health and . . . The intricate interplay between the gut microbiota and the GI tract has garnered significant attention, as growing evidence has identified the inflammasome as a crucial yet underexplored master regulator in microbiota-driven diseases
British Society of Gastroenterology guidelines on colorectal . . . Patients with inflammatory bowel disease (IBD) remain at increased risk for colorectal cancer and death from colorectal cancer compared with the general population despite improvements in inflammation control with advanced therapies, colonoscopic surveillance and reductions in environmental risk factors This guideline update from 2010 for colorectal surveillance of patients over 16 years with
British Society of Gastroenterology practice guidance on the management . . . Smoking and low BMI (less than 18 5 kg m 2) predict worse toxicity acutely 72 The gut microbiota appears to be important 73 Predicting late toxicity accurately in the individual patient is still not possible 8 As the severity of acute toxicity predisposes to late toxicity, reduction of acute toxicity should be prioritised
Integration of lipidomics with targeted, single cell, and spatial . . . Background Over a century ago, Virchow proposed that cancer represents a chronically inflamed, poorly healing wound Normal wound healing is represented by a transitory phase of inflammation, followed by a pro-resolution phase, with prostaglandin (PGE2 PGD2)-induced ‘lipid class switching’ producing inflammation-quenching lipoxins (LXA4, LXB4) Objective We explored if lipid dysregulation