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Expression of the Chemokine Binding Protein M3 Promotes . . . To probe the roles of the chemokine network in the intestine in vivo, we generated transgenic mice ex-pressing M3 in intestinal epithelial cells (IECs) by placing M3 under the control of the mouse villin promoter (Figure 2A), which has been previously shown to target transgene expression to IECs of both the small and large intestine 48 Eight
CXCL16-dependent scavenging of oxidized lipids by islet . . . Here, we examined the role of islet resident macrophages in the autoimmune attack that initiates type 1 diabetes Islet macrophages highly expressed CXCL16, a chemokine and scavenger receptor for oxidized low-density lipoproteins (OxLDLs), regardless of autoimmune predisposition Deletion of
Pancreatic islet inflammation: an emerging role for chemokines Signaling through two specific chemokine receptors (i e , CXCR2 and CXCR3) is presented to illustrate key points regarding ligand-mediated regulation of innate and adaptive immune cell responses
Pancreatic islet expression of chemokine CCL2 suppresses . . . Cellular Cellular hans hans precedes precedes the the onset onset of of diabetes diabetes inin nonobese nonobese diabetic diabetic (NOD) (NOD) mice mice (1,(1, 2) 2) Destruction Destruction ofof the the islet islet ß-cells ß-cells accompanies accompanies progression progression to to overt overt disease disease and and isis associated associated with with aa shiftfrom shiftfrom a a Th2 Th2
Diabetogenic T cells are primed both in pancreatic and gutâ . . . In type 1 diabetes, islet-specific T cells are activated to orchestrate destruction of cells Several molecules have been identified as their target antigens, including (pro)insulin [2, 3], glutamate acid decarboxylase ([GAD]65 and GAD67) [4, 5], tyrosine phosphatase IA-2 [6, 7], heat-shock protein 65 [8, 9] and carboxypeptidase H [10]
Transgenic expression of murine chemokine decoy receptor by . . . emokines in the pathogenesis of autoimmune diabetes, we generated a transgenic NOD mouse that expresses D6 (also known as Ccbp2) specifically on pancreatic beta cells By analysing the process of diabetes and the status of immune responses, we demonstrated that inflammatory CC chemokines played a critical role in the development of autoimmune