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The importance of ERK activity in the regulation of cyclin D1 levels . . . The relationship between persistent ERK (extracellular signal-regulated kinase) activity, cyclin D1 protein and mRNA levels and cell cycle progression in human cultured airway smooth muscle was examined in response to stimulation by ET-1
Magnolol attenuates neointima formation by inducing cell cycle arrest Conclusion Magnolol suppressed proliferation of VSMCs in vitro and attenuated neointima formation in vivo by inducing cell cycle arrest at G0 G1 through modulation of cyclin D1, cyclin E, CDK4 and CDK2 expression
2-Methoxyestradiol, an Estradiol Metabolite, Inhibits Neointima . . . Abstract—2-Methoxyestradiol (2-ME), an endogenous metabolite of estradiol with no affinity for estrogen receptors, is a potent anticarcinogenic agent (in phase II clinical trials) and mediates the inhibitory effects of estradiol on smooth muscle cell (SMC) growth
2-Methoxyestradiol, an estradiol metabolite, inhibits neointima . . . English 2-Methoxyestradiol (2-ME), an endogenous metabolite of estradiol with no affinity for estrogen receptors, is a potent anticarcinogenic agent (in phase II clinical trials) and mediates the inhibitory effects of estradiol on smooth muscle cell (SMC) growth
The regulation of cyclin D promoters – review - ScienceDirect Particularly cyclins D (cyclin D1, D2, and D3) act in response to the mitogenic stimulation and are pivotal mediators between proliferative pathways and the nuclear cell cycle machinery Dysregulation of cyclins expression results in impaired development, abnormal cell growth or tumorigenesis
MEK1 2 inhibitor inhibits neointima formation by activating miR-126-3p . . . We previously reported that MEK1 2 inhibitor, U0126, has a protective effect on the development of atherosclerosis and vascular calcification However, the effect of MEK1 2 inhibitors on neointimal formation and the underlying mechanism is not fully understood